I won't disguise my animosity toward Hillary Clinton. But I am presenting here, for lay person's understanding, the neurologic disorders exhibited on video from multiple sources at multiple times. Extensive literature research has gone into the production of this essay, which will no doubt be labelled as fantasy by Clintonites. However the sources are independent, international and appearing in medically peer-revised journals. After talking to a very intelligent gentleman with a post-graduate degree, it became apparent to me that what I thought was obvious, was opaque to him. The problem is that he doesn't speak "medical". Be warned that I am fully aware that the main readership of this blog is non-medical. Therefor, I will make every effort to write in English instead of medical. Suffice it to say, that since I have only at work on this project for 5 days, I have reviewed over 50 journal articles. The reference to these articles are not intended for a lay audience, but the citations are presented just so that the factual basis for my assertions will be researchable by anyone who has the interest. Furthermore, I welcome questions in the comment thread (though I am not always prompt to respond to them. Anyone interested in more information, including URLs for the medical articles can c99-mail me.

Due to the subject, Hillary Clinton, we may assume several things, which are not medical but political in nature.
1. You cannot believe anything she says
2. We do not have access to her medical records, about which I will comment later
3. None of the so-called medical experts commenting on her condition did not have access to her medical records

As a consequence I feel it is incumbent upon her to release her medical records promptly.

The reason why I undertook this project was because of a video taken as she was walking off the stage following her acceptance speech. I then checked that reliable medical source YouTube (snark) for more videos. And there are quite a few. I have only seen 4 which I consider definitely neuropathological but there are probably more.

There are several other issues here about this essay. The list of references is not complete, as I felt that for the purposes of this essay that would be overkill. A second point also relates to listing my sources (references) is that this prospect has been entirely more time-consuming than I first envisioned, hence the references are not listed alphabetically by the first authors's last name.

Before we get to the video analysis, let's discuss what we do know about what happened to her in December, 2012. Alas, we know very little since, of course the reporting was done almost exclusively by the Main Stream Media (MSM).

First reportage in print of which I am aware is from CNN on December 15, 2012. The other almost contemporary report is from ABC news. the next press report which I found pertinent (which is not to say that my review of MSM offerings was comprehensive) came from CNN also.

On January 1, while Clinton was checking out of the hospital, further information relating to her medical condition was released

Mrs. Clinton, 65, was admitted to NewYork-Presbyterian/Columbia hospital on Sunday after a scan discovered the blood clot. The scan was part of her follow-up care for a concussion she sustained more than two weeks earlier, when she fainted and fell, striking her head.

. The citation for this is:
http://www.nytimes.com/2013/01/03/us/politics/hillary-clinton-is-dischar...

From this very limited sourcing, we have learned two things, which are unchallenged by Clinton:
1. She had a head injury resulting in concussion.
2. She sustained a symptomatic right lateral sinus thrombosis about two weeks later.
That information is undisputed.

Then there follows just two of the videos posted on YouTube
1. https://www.youtube.com/watch?v=YMHOcmDVBP0 Hillary tries to laugh off seizure

This video demonstrates several things, best seen with slow motion:
Her eyes look down and to the right along with slight head turning briefly; immediately thereafter her head returns to neutral position but her eyes are not well-visualized because of slow blinking, slower than normal. Immediately thereafter, rapid neck flexion-extension oscillations begin; some brief time afterward she begins laughing but not making eye contact initially. These are involuntary movements--in other words, she had no control over them. Later, I will give scientific citations to what I am now proposing: first, this could be a manifestation of gelastic epilepsy, often seen with medial temporal lobe but more often frontal lobe seizures. Gelastic means "laughing". Secondly, but less likely this could be a manifestation of torticollis, which means "twisted neck". The problem with that explanation is that emotional displays do not occur. Third, it could be an immensely clumsy attempt at humorous interaction. This third alternative is also unlikely, because in previous video imaging of her laughing (or cackling) there are no such untoward movements.

2. https://www.youtube.com/watch?v=OqbDBRWb63s, the truth about Hillary's Bizarre Behavior.
First of all, ignore 90% of what the narrator says. He seems to have little understanding of the issues he discusses. Example, her laughter (other than the subject of the preceding video) is not psychotic. Plus the narrator conflates two conditions: personality disorder, which develops early in life and is not evidence of brain injury. Some of the tendencies of her sociopathic/narcissistic traits would be ease of irritation, explosive bad temper (sometimes violent), and impulsivity. Consult Diagnostic and Statistical Manual, edition 5 for descriptions of the various personality disorder.
Now, having said that, let me add that either or one or both of her known medical conditions could exacerbate her psychopathic traits.

The key video here is the one obtained after the nomination acceptance speech. Again, slow motion tells the best story because important finding occurred but too briefly to be detected at normal projection speed. In the video, while walking off stage, Hillary's eyes open widely, staring straight ahead while her jaws become slack and open--this is fleeting. What happens next is diagnostic of focal right frontal lobe epilepsy: she turns here eyes which are wide open upward to the left, turns her head upwards to the left, opens her mouth widely--but notice it opens more widely on the left side than the right. These are typical signs of an aversive seizure in which the body parts move in the opposite side to the side of the brain which is abnormally excited.

Mild head trauma, by which the lay audience understands as concussion with brief loss of consciousness, actually spans a spectrum varying from no loss of consciousness at all, to prolonged deep coma. Focal neurological signs may or may not be present. Intracranial bleeding may occur even in the absence of unconsciousness. Headache, although present in about 90% of concussion, may be totally absent, or quite transient, or severe, prolonged and disabling. Due to the varying circumstances associated with concussion, there is great heterogeneity of pathologic responses both in location and severity.

Mild traumatic brain injury (mTBI) is common in the United States, with estimates ranging between 1.3 million and 1.7 million per year. Of those TBIs, approximately 75-80% are mTBI. Only a small portion of otherwise neurologically intact are hospitalized unless acute imaging shows a lesion. Other reasons for hospitalization are failure of sensorium to clear, persistence or development of neurological signs or worsening headaches despite normal imaging.

Although the majority of patients with uncomplicated mTBI recover completely, there is a small but significant minority who do not. Other than outright neurological deficits, the bane of mTBI research and treatment has been the "post-concussion syndrome" (PCS). Only within the past 10 years has real progress been made between discerning purely psychological symptomatology and validated proof of underlying neuropathology. Five percent of mTBI patients go on to have PCS lasting 1 year or more.

This non-technical article describes common perceptions about minor head injury

This highly technical article discusses specific brain architecture changes following mTBI accompanied by PCS.

This article deals with neuropsychologic measures of PCS but no anatomic correlation with imaging. It contains a good description of some of the most commonly utilized neuropsychologic tests.

This article demonstrates early anatomic brain changes following MVA as demonstrated with advanced neuroimaging techniques within days of MVA. Most of the MVAs invalid neck extension-flexion injuries but some presumably were rotatory. Just as in concussion as a general field, MVA related neurotrauma is heterogeneous.

This article is long and complex but it illustrates very well the subtle anatomicdisturbances which are technically caused lesions. Though complex, these studies are totally safe (providing the patient has no metallic implants other than certain MRI-safe orthopedic implants), painless and noninvasive.

This citation is about an MRI-based technique which objectively quantifies chronic mTBI

Here is a very salient citation, concerning intermittent involuntary emotional outbursts, previously known as
pseudobulbar affect of which the primary symptoms include emotional volatility, pathological laughter--or crying. One of the causes for this condition is mTBI.

Another very appropriate question is whether Hillary Clinton is aware of her peculiar (epileptic) behavior. Surprisingly, the answer is No.

Saving the best for last: manifestations of frontal lobe seizures. Although the seizures described herein are "intractable". the general description of individual seizures comports with the video imaging and the medical literature cited not only in this essay but in the international literature. Note that some epileptics experienced coughing fits.

Commentary
Please consider this a work in progress. There is much more information to be explained by me--and I hope by others. Some of those issues involve further exploration of the post-concussion syndrome, neuropathology of cerebral dural sinus thrombosis, and inter-relations between post-concussion syndrome and cerebral damage from dural sinus thrombosis.

Admittedly, some of my remarks may be considered speculative. It would be delightful if knowledgable neuroscientists would comment on this work.

I make two requests of you, my fellow c99ers:
1. Please be liberal in your comments. Send me c99-mail questions/comments as well.
2. SPREAD THE WORD.